Download Atypical Antipsychotics (Medical Psychiatry) by John C. Csernansky, John Lauriello PDF

By John C. Csernansky, John Lauriello

Showcasing the newest reports within the box, this reference unveils fresh breakthroughs within the use of odd antipsychotics for the therapy of numerous sufferer populations-tracking advancements within the administration of sufferers with schizophrenia and affective psychotic problems, in addition to healing regimens for kids and young people.

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Extra info for Atypical Antipsychotics (Medical Psychiatry)

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Antipsychotic-induced weight gain: a comprehensive research synthesis. Am J Psychiatry 1999; 156(11):1686–1696. 102. Wirshing DA, et al. Novel antipsychotics: comparison of weight gain liabilities, J Clin Psychiatry 1999; 60(6):358–363. 103. Schlicker E, Marr I. The moderate affinity of clozapine at H3 receptors is not shared by its two major metabolites and by structurally related and unrelated atypical neuroleptics. Naunyn Schmiedebergs Arch Pharmacol 1996; 353(3):290–294. 104. Kathmann M, Schlicker E, Gothert M.

Bodyweight gain with atypical antipsychotics. A comparative review. Drug Saf 2001; 24(1):59–73. 10. McIntyre RS, McCann SM, Kennedy SH. Antipsychotic metabolic effects: weight gain, diabetes mellitus, and lipid abnormalities. Can J Psychiatry 2001; 46(3):273–281. 11. Fontaine KR, et al. Estimating the consequences of anti-psychotic induced weight gain on health and mortality rate. Psychiatry Res 2001; 101(3):277–288. 12. Stip E. Novel antipsychotics: issues and controversies. Typicality of atypical antipsychotics.

ATYPICAL ANTIPSYCHOTICS AND DEPOLARIZATION BLOCK OF DOPAMINE NEURONS As stated above, all antipsychotic drugs, typical and atypical block DA receptors. Although not without controversy, this action is believed to result in the eventual inactivation of DA neurons in the mesolimbic and mesocortical projections (41). This effect, however, is only acquired following a relatively prolonged exposure to the drug (2 weeks or longer). Thus, this is an attractive mechanism to explain the clinical effects of APDs, since the therapeutic actions of APDs do not match the time course of postsynaptic DA receptor blockade, but do match the time course of these delayed effects on presynaptic activity (42).

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